. Dossier international de la médecine . et surtout pour comparaison avec les drawingsappartenant aux sections II et IV—nous avons reproduit les plaques originales accompagnant les articles analysés dans cette section. XO. Cas de Kahler et de Pick (22), 1879.—l'auteur a hésité à classer ce cas parmi les cas erronés de malformations de la moelle épinière, mais il a été inclus avec eux, parce qu'il semble diflicult d'exclure la disfacilité—tabes dorsalis—comme le facteur produisant les changesdécrits par les auteurs comme une malformation. Dans ce cas, la malformation consistait en une anomalie de l'étroitesse

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. International record of medicine . and especially for comparison with the drawingsbelonging to Sections II and IV—we have reproduced theoriginal plates accompanying the articles analyzed in thissection. Xo. 1. Case of Kahler and Pick (22), 1879.—The writerhas hesitated about classing this case among the erroneous instances of spinal-cord malformations, but it has been in-cluded with them, because it seems diflicult to exclude dis-ease—tabes dorsalis—as the factor producing the changesdescribed by the authors as a malformation. The malformation in this case consisted in an abnormalnarrowness or thinning of the posterior horns, so that thecontour of the gray horns resembled the type seen in ani-mals, such as the ungulate or carnivorous orders, rather thanthe classical type characteristic of the human cord. (SeeFig. 1.) The posterior or white columns were also verynarrow, and the columns of Clarke appeared to be defectivein the middle dorsal region, although normal in the lowestdorsal and uppermost lumbar regions.. Fig. 1.—Kahler and Picks case. The cord came from a case of tabes, and showed theusual lesion in the posterior columns. The authors, how-ever, dismiss the question of the production of the changein the posterior horns by the tabetic lesion in the posteriorwhite columns, because these were not retracted at theirperipheral margins, and the tissues of the narrowed pos-terior horns did not show the structure of the old andshrinking stage of sclerosis. But these are not veryconvincing reasons for rejecting the tabetic origin of thedeformity. After dismissing the tabetic origin of the deformity, the view is presented that the change in the shape of theposterior horns was a congenital malformation due to a de-ficient embryonal site [Anlage) or development of the pos-terior columns or horns. The suggestion is finally madethat the primary starting point in this defective develop-ment of the cord was localized in a deficient volumetricfoetal arrangement or

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